Amyotrophic lateral sclerosis (ALS)

Responsible pathogen complex #

Amyotrophic lateral sclerosis reflects progressive neuromuscular degeneration associated with a chronic infectious and parasitic burden that impairs liver detoxification, pancreatic regulation, vascular integrity, and systemic immune stability. Rather than isolated motor neuron decline, persistent microbial stress interferes with metabolic and neurological resilience.

Liver and pancreatic flukes play a central systemic role.

Fasciola hepatica

Impairment of bile flow and hepatic detoxification increases circulating toxins and inflammatory mediators that affect neural tissue stability.

Clonorchis sinensis and Fasciolopsis buski

Chronic fluke burden sustains immune activation and interferes with nutrient absorption, contributing to metabolic stress that weakens neuromuscular integrity.

Eurytrema pancreaticum

Pancreatic involvement disrupts metabolic regulation and endocrine balance, influencing neuromuscular energy supply.

Intestinal and systemic bacterial burden intensifies inflammatory signaling.

Shigella

Associated with toxin production and systemic immune activation may influence neurological inflammation.

Nocardia asteroides

Persistent organisms are capable of long-term immune stimulation and tissue stress.

Additional flukes contribute to structural and vascular strain.

Echinostoma revolutum, Echinoparyphium recurvatum, and Gastrothylax elongatus

Parasitic organisms that maintain chronic antigen load and inflammatory signaling, affecting systemic resilience.

Streptococcal and staphylococcal species reinforce chronic immune activation.

Streptococcus species

Sustains inflammatory responses and may contribute to ongoing immune-mediated tissue stress.

Staphylococcus aureus

Produces toxins that intensify systemic inflammation and vascular irritation.

Integrated impact

Parasitic burden impairs detoxification and metabolic regulation. Bacterial pathogens sustain inflammatory signaling. Chronic immune activation interferes with neuromuscular stability. Together, this pathogen complex supports progressive motor weakness and neurological degeneration characteristic of ALS.

Frequency session table #

Pathogen	Use freq. (kHz)	Freq. low–high (kHz)
Fasciola hepatica	423	421.35–427.3
Fasciola hepatica cercariae	425	423.8–430.6
Fasciola hepatica eggs	427	422.0–427.6
Fasciola hepatica miracidia	—	421.75–424.7
Fasciola hepatica rediae	—	420.6–427.5
Clonorchis sinensis	427	425.7–428.75
Fasciolopsis buski adult	434	427.7–435.1
Fasciolopsis buski eggs	434	427.35–435.45
Fasciolopsis buski (2nd range)	432	429.5–436.25
Fasciolopsis buski (3rd range)	—	427.35–435.2
Fasciolopsis buski (4th range)	—	427.3–433
Eurytrema pancreaticum	421	420.35–422.3
Shigella dysenteriae	390.089	390.089–390.089
Shigella flexneri depression	394	394–394
Shigella sonnei invades tumors	318	318–318
Nocardia asteroides found in Parkinson’s Disease	355.1	354.95–355.35
Nocardia asteroides (2nd range)	368	363.7–370
Echinostoma revolutum	428	425.5–429.65
Echinoparyphium recurvatum	421	418.55–423.9
Gastrothylax elongatus	455	451.9–457.1
Streptococcus lactis occurs in milk	385	382–387
Streptococcus mitis lung infection, tooth infection, abscesses, causes stiff knees	318	313.8–321.1
Streptococcus pneumoniae causes pneumonia and inner ear disease	368	366.85–370.2
Streptococcus pyogenes (tooth)	373	369.5–373.0
Staphylococcus aureus (culture)	378	376.27–380.85
Staphylococcus aureus (slide) source is tooth infection, causes abscesses, heart disease, invades tumors	381	381–381