Crohn’s disease

Responsible pathogen complex #

Crohn’s disease develops within a chronically inflamed intestinal environment sustained by multiple interacting pathogens. Rather than a single infectious agent, a layered microbial and toxic burden weakens barrier integrity, overloads detox pathways, and maintains immune overactivation in the bowel.

Shigella species

These bacteria directly irritate and penetrate the intestinal lining. Their toxins increase permeability and provoke persistent inflammatory signaling within the gut wall.

Salmonella species

Release endotoxins that sustain immune stimulation. They disturb microbial equilibrium and reinforce chronic intestinal inflammation.

Fasciola hepatica

This liver fluke burdens bile ducts and reduces detox efficiency. Impaired bile flow alters intestinal microbial control and promotes ongoing irritation.

Fasciolopsis buski

Resides in the intestine and creates chronic mucosal stress. Its presence supports bacterial persistence and mechanical irritation.

Clonorchis sinensis

Occupies bile structures and contributes to congestion. Reduced bile regulation affects digestion and increases inflammatory susceptibility.

Eurytrema pancreaticum

Interferes with pancreatic enzyme secretion. Incomplete digestion leads to fermentation and further irritation of intestinal tissue.

Escherichia coli

When overrepresented, produces endotoxins that intensify immune activation and damage the mucosal barrier.

Ascaris

Migratory parasite that transports microbes and toxic residues through tissues, increasing systemic inflammatory load.

Candida albicans

Opportunistic yeast that overgrows in weakened intestinal terrain. Produces irritative metabolites that destabilize the gut barrier.

Aflatoxin and Sterigmatocystin

Fungal toxins that add chemical stress to already inflamed tissues and impair cellular repair.

Endamoeba gingivalis trophozoite and Endolimax

Protozoan organisms contributing additional microbial pressure and immune stimulation.

Functional impact of the complex

The combined burden of parasites, bacteria, protozoa, and fungal toxins weakens intestinal defenses and sustains chronic immune activation. Removing these infectious and toxic barriers restores stability to the bowel environment and allows mucosal repair mechanisms to function effectively.

Pathogen / Toxin	freq. (kHz)	Freq. low–high (kHz)
Shigella dysenteriae intestinal problems	390.089	390.089–390.089
Shigella flexneri depression	394	394–394
Shigella sonnei invades tumors	318	318–318
Salmonella enteriditis intestinal infection	329	329–329
Salmonella paratyphi	368	365.05–370.1
Salmonella typhimurium food poisoning, nervousness, apathy	385	382.3–386.55
Salmonella typhimurium food poisoning, nervousness, apathy (2nd freq.)	355	—
Salmonella typhimurium food poisoning, nervousness, apathy (3rd freq.)	386	—
Salmonella typhimurium food poisoning, nervousness, apathy (4th freq.)	390	—
Fasciola hepatica adult	423	421.35–427.3
Fasciola hepatica adult (2nd freq.)	425	423.8–430.6
Fasciola hepatica adult (3rd freq.)	427	422.0–427.6
Fasciola hepatica adult (4th range)	—	421.75–424.7
Fasciola hepatica adult (5th range)	—	420.6–427.5
Eurytrema pancreaticum adult	421	420.35–422.3
Clonorchis sinensis adult	427	425.7–428.75
Fasciolopsis buski adult	432	427.7–435.1
Fasciolopsis buski adult (2nd freq.)	434	427.35–435.45
Fasciolopsis buski adult (3rd range)	—	429.5–436.25
Fasciolopsis buski adult (4th range)	—	427.35–435.2
Fasciolopsis buski adult (5th range)	—	427.3–433
Escherichia coli	356	356–356
Escherichia coli (2nd freq.)	393	392–393
Ascaris larvae in lung common roundworm of cats and dogs	408	404.9–409.15
Ascaris megalocephala (male)	408	403.85–409.7
Candida albicans	386	384.2–388.4
Aflatoxin	177	177–177
Aflatoxin (2nd freq.)	188	177–177
Aflatoxin (3rd freq.)	133	133–133
Endamoeba gingivalis trophozoite	438	433.8–441
Endolimax nana trophozoites and cysts (2nd range)	—	430.5–433.5