Cysts

Responsible pathogen complex #

Cysts represent the body’s attempt to isolate something it cannot immediately eliminate. When parasites invade tissue and are accompanied by bacteria, viruses, and toxic byproducts, the immune system may surround the affected area with connective tissue. This containment strategy prevents spread but also creates a persistent structural abnormality. Successful resolution requires addressing the entire constellation of pathogens rather than focusing solely on the cyst.

Fasciola hepatica

This liver fluke, including its cercariae, eggs, miracidia, and rediae stages, is central in many chronic tissue disturbances. Its migration and presence in organs provoke inflammation and obstruction. When fluke stages remain embedded, tissue encapsulation can occur, forming cystic structures.

Clonorchis sinensis and Fasciolopsis buski

These flukes and their developmental stages add further parasitic burden. Adult forms irritate ducts and soft tissue. Eggs and larval stages maintain ongoing immune activation. The recurring cycle of irritation and repair promotes fibrotic walls around infected areas.

Eurytrema pancreaticum

This pancreatic fluke affects glandular tissue. Chronic irritation of ducts and secretory cells may lead to localized swelling and encapsulation.

Adenovirus

Viruses such as Adenovirus interfere with normal cellular regulation. When viral activity accompanies parasitic infestation, cellular repair becomes disorganized, increasing the likelihood of the formation of abnormal tissue pockets.

Gardnerella vaginalis, Chlamydia trachomatis, Neisseria gonorrhea, Treponema pallidum, and Trichomonas vaginalis

These genital tract pathogens promote chronic low-grade infection. Persistent inflammation in reproductive tissue creates an environment conducive to cyst development, particularly when drainage pathways are obstructed.

Salmonella species, Campylobacter pyloridis, and Escherichia coli

Intestinal bacteria can migrate or affect adjacent organs through toxin production. Their metabolic byproducts stress liver and lymphatic clearance, indirectly contributing to tissue stagnation and cyst formation.

Proteus mirabilis and Proteus vulgaris

These urinary tract pathogens cause recurring irritation and a toxic burden. When detoxification is impaired, tissue encapsulation becomes a defensive strategy.

Clostridium species

Toxin-producing anaerobes such as Clostridium acetobutylicum, Clostridium perfringens spores, Clostridium septicum, and Clostridium botulinum disturb cellular respiration and local immunity. Toxin exposure promotes inflammatory pockets that may wall off into cystic formations.

This frequency program systematically targets parasites, associated microbes, and their toxic influences. By removing the irritative stimulus and reducing microbial load, it dismantles the conditions that maintain cyst walls. Once the underlying burden is cleared, normal tissue remodeling and drainage can resume, allowing the body to resolve the structure naturally rather than merely containing it.

Frequency session table #

Pathogen	Use freq. (kHz)	Freq. low–high (kHz)
Fasciola hepatica	423	421.35–427.3
Fasciola hepatica cercariae	425	423.8–430.6
Fasciola hepatica eggs	427	422.0–427.6
Fasciola hepatica miracidia	—	421.75–424.7
Fasciola hepatica rediae	—	420.6–427.5
Clonorchis sinensis	427	425.7–428.75
Fasciolopsis buski adult	434	427.7–435.1
Fasciolopsis buski adult (2nd freq)	432	—
Fasciolopsis buski eggs	—	427.35–435.45
Fasciolopsis cercariae	—	429.5–436.25
Fasciolopsis miracidia	—	427.35–435.2
Fasciolopsis rediae	—	427.3–433.0
Eurytrema pancreaticum	421	420.35–422.3
Adenovirus	393	393–393
Adenovirus (2nd range)	—	371.45–386.9
Gardnerella vaginalis ovarian and genital tract infection	340	338.0–342.55
Treponema pallidum causes syphilis	347	346.85–347.4
Neisseria gonorrhea causes gonorrhea	334	333.85–336.5
Salmonella enteriditis intestinal infection	329	329–329
Salmonella paratyphi	368, 385	365.05–370.1
Salmonella typhimurium food poisoning, nervousness, apathy	355, 386, 390	382.3–386.55
Salmonella typhimurium (2nd freq.)	386	—
Salmonella typhimurium (3rd freq.)	390	—
Campylobacter fetus smear	368	365.3–370.6
Campylobacter pyloridis	355	352.0–357.2
Escherichia coli (E. coli) intestinal bacterium	356, 393	356–356
Escherichia coli (E. coli) (2nd range)	—	392–393
Proteus mirabilis	324, 349	320.55–326.0
Proteus mirabilis (2nd range)	—	345.95–352.1
Proteus vulgaris urinary tract “urinary tract” pathogen	413, 336, 328	408.75–416.45
Proteus vulgaris (2nd range)	—	333.75–339.15
Proteus vulgaris (3rd range)	—	327.2–329.5
Chlamydia trachomatis	381	379.7–383.95
Trichomonas vaginalis	381	378.0–383.6
Clostridium acetobutylicum	389, 384	382.8–391.15
Clostridium botulinum (tooth)	362	361.0–364.55
Clostridium perfringens spores	396	394.2–398.1
Clostridium septicum	364	362.05–365.6