Menopausal dysfunction

Responsible pathogen complex #

Menopausal dysfunction reflects chronic infectious and parasitic pressure affecting liver metabolism, reproductive tissues, intestinal balance, and pancreatic regulation. Rather than a purely age-related hormonal decline, persistent microbial burden interferes with estrogen metabolism, vascular stability, and endocrine coordination.

Genital pathogens sustain pelvic inflammatory stress.

Gardnerella vaginalis ovarian and genital tract infection

Alters reproductive tract microbiota and maintains mucosal irritation. Chronic imbalance destabilizes local hormone-responsive tissues.

Chlamydia trachomatis, Trichomonas vaginalis, Treponema pallidum, and Neisseria gonorrhea causes gonorrhea

Persistent urogenital infections capable of long-term survival. They sustain inflammatory signaling and impair tissue perfusion within pelvic organs.

Enteric and urinary bacteria reinforce systemic immune activation.

Proteus species, Salmonella species, Campylobacter species, and Escherichia coli

Chronic intestinal and urinary tract colonization maintains cytokine activity and toxin exposure. This persistent immune burden interferes with hepatic hormone processing and vascular stability.

Liver and pancreatic flukes disturb endocrine metabolism.

Fasciola hepatica

Impairs bile flow and hepatic detoxification. Disrupted estrogen clearance contributes to vasomotor instability and hormonal fluctuation.

Clonorchis sinensis and Fasciolopsis buski

Flukes affecting biliary and intestinal tissues. Their presence reinforces chronic immune activation and metabolic imbalance.

Eurytrema pancreaticum

Pancreatic fluke that interferes with metabolic regulation and endocrine signaling, contributing to irregular hormonal adaptation during menopause.

Integrated impact: Chronic pelvic infection damages local tissue stability. Enteric bacteria sustain inflammatory signaling. Liver and pancreatic parasites impair hormone metabolism and clearance. Together, this pathogen complex contributes to vasomotor symptoms, mood fluctuation, metabolic instability, and persistent menopausal discomfort.

Frequency session table #

Pathogen	Use freq. (kHz)	Freq. low–high (kHz)
Gardnerella vaginalis ovarian and genital tract infection	340	338–342.55
Proteus mirabilis	324	320.55–326
Proteus mirabilis (2nd freq.)	349	345.95–352.1
Proteus vulgaris urinary tract infection	413	408.75–416.45
Proteus vulgaris (2nd freq.)	336	333.75–339.15
Proteus vulgaris (3rd freq.)	328	327.2–329.5
Salmonella enteriditis intestinal infection	329	329–329
Salmonella paratyphi	368	365.05–370.1
Salmonella typhimurium food poisoning, nervousness, apathy	385	382.3–386.55
Salmonella typhimurium food poisoning, nervousness, apathy (2nd use freq.)	355	—
Salmonella typhimurium food poisoning, nervousness, apathy (3rd use freq.)	386	—
Salmonella typhimurium food poisoning, nervousness, apathy (4th use freq.)	390	—
Campylobacter fetus smear	368	365.3–370.6
Campylobacter pyloridis	355	352–357.2
Chlamydia trachomatis	381	379.7–383.95
Treponema pallidum	347	346.85–347.4
Neisseria gonorrhea causes gonorrhea	334	333.85–336.5
Trichomonas vaginalis	381	378–383.6
Escherichia coli (E. coli) intestinal bacterium	356	356–356
Escherichia coli (E. coli) intestinal bacterium (2nd freq.)	393	—
Fasciola hepatica	423	421.35–427.3
Fasciola hepatica cercariae	425	423.8–430.6
Fasciola hepatica eggs	427	422–427.6
Fasciola hepatica miracidia	—	421.75–424.7
Fasciola hepatica rediae	—	420.6–427.5
Clonorchis sinensis	427	425.7–428.75
Fasciolopsis buski adult	434	427.7–435.1
Fasciolopsis buski eggs	434	427.35–435.45
Fasciolopsis buski (2nd range)	432	429.5–436.25
Fasciolopsis buski (3rd range)	—	427.35–435.2
Fasciolopsis buski (4th range)	—	427.3–433
Eurytrema pancreaticum	421	420.35–422.3